Dr. Wolfgang Lutz: Figures from "Dismantling a Myth", Chapter VII, page 125-180


Page 149 - Fig. 16:
103 patients suffering from Crohn's disease were treated by a low-carbohydrate diet. After a quarter of year most patients (85 percent) showed remarkable improvement in their health. After half a year, more than 60 percent were asymptomatic, after one year more than 70 percent and after one and a half year about 85 percent. This is in contrast with ulcerose colitis, which Is shown in the lowest line, improvement of which runs much slowlier on the same diet and often is interrupted by relapses.

Crohn's disease generally thought of being incurable can so be shown to be very well accessable to dietary measures.




protein fat carbohydrate kcal kilojoule
sum of 12 cases 1241 1866 868 25422 106391
average per case 103 156 72 2118 8866
kcal per 24 hours 422 1395 295 - -
Percent calories of main
nutrient sources
20 66 14 - -


Table 1:
Values for actually consumed nutrient components of twelve patients with ulcerative colitis after a low-carbohydrate diet for several months.




Absolute
Percent
years 0 2 4 6 8 <8
number of patients 74 74 54 46 35 28
without findings - 40 41 39 32 26
contact hemorrhaging 24 19 6 4 2 1
spontaneous hemorrhaging 23 7 4 2 1 -
ulcers and fibrin 26 4 3 2 - 1
years 0 2 4 6 8 <8
number of patients 100 100 100 100 100 100
without findings - 54 76 85 91 93
contact hemorrhaging 32 26 11 9 6 4
spontaneous hemorrhaging 31 10 7 4 3 -
ulcers and fibrin 36 5 6 4 - 4


Table 2:
Progress of ulcerative colitis of 74 patients' on a low-carbohydrate diet. Criteria: rectoscopic findings. Decline of hemorrhage and ulcers during two to eight years. Increase of rate of healing to over 90 percent.





Page 155 - Fig. 11:
Graphic depiction of the values of table 2. The most severe symptoms disappear first, contact hemorrhaging last. Simultaneous increase of percentages of symptom-free patients
_____   = without findings;
. . . .   = only contact hemorrhaging;
- - -   = spontaneous hemorrhaging;
- . - .   = ulcers and fibrin




Page 162 - Fig. 18:
The behaviour of hypo- (a) and hypersideroses (b) on a low-carbohydrate diet (72 g) without other therapeutic measures.

a: 38 cases with manifest hyposiderosis. In in average a normal iron level is attained after six months.

b: 38 cases with hypersiderosis approaching normal within then weeks. The main effect of the carbohydrate restriction is seen in the first four weeks, in which the mean value drops to 140 milligram. The figures of "n =" show how many pations were investigated at each point.




page 166 - Fig. 19:
Abnormal low calcium levels normalize with carbohydrate restriction.




Page 172 - Fig. 20:
The behaviour of GOT (ASAT), a parameter indicative of the state of the liver. The speed with which a normal value is obtained on a low-carbohydrate diet depends upon the initial value (A,B,C) . Even in severe cases (C) the results are impressive.
n = number of cases Investigated at each date.




Page 173 - Fig. 21:
The LAP level (leucine-aryl amidase) is characteristic of the function of the bile capillaries and the unimpeded flow of bile. Even elevated LAP values rapidly returned to normal on a low-carbohydrate diet.




Page 174 - Fig. 22:
Chronically elevated serurn bilirubin levels are generally considered to be incurable. Observation of such patients on a low-carbohydrate diet, however, shows that even high values for bile pigments in the blood can be lowered.
n = number of cases investigated at each date.




Page 175 - Fig. 23:
GammaglobuIin as percent of totaI protein in a case of liver cirrhosis with ascites. The rise on a low-carbohydrate diet reflects the general anabolic effect of the diet and indicates the formation of antibodies. This is an example of a detrimental effect not planned by nature.







ISBN 3-9215O0-24-9


These figures are from "Dismantling a Myth - The Role of Fat and Carbohydrates in our Diet" by Dr. Wolfgang Lutz.
© 1986 by Selecta-Verlag
Dr. Ildar Idris GmbH & Co. KG Planegg V. Munchen, West Germany.
Original title "Leben ohne Brot", translated by Beatrice Idris-Duncan and Joy Wieser.

All rights reserved. No part of this book may be reproduced in any form or by any means electronic or mechanical, including photocopying, recording, or by any information storage and retrieval system, without permission in writing from the publisher.





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